by Rebecca Prade | Jan 31, 2026 | NUTRITION
Magnesium has been part of the wellness conversation for years, but new 2025 research has positioned it firmly within the longevity space. A recent scoping review published in Nutrients links long-term magnesium intake with better healthspan and reduced mortality risk. The takeaway is simple: magnesium isn’t just a calming mineral for evening routines. It underpins fundamental systems that shape how well we age.
Why magnesium matters more than we realised
Magnesium is involved in more than 300 enzymatic reactions. In clinic, low or borderline magnesium is one of the most common patterns I see in women struggling with energy, sleep, mood, stress resilience, bloating or muscle tension. Levels may still fall within a normal range on blood tests, but functionally the body is working harder without enough of this mineral.
The new longevity data strengthens this picture
The magnesium depletion score
The Nutrients review introduced a tool called the magnesium depletion score, designed to estimate long-term risk by assessing factors that reduce magnesium status over time: alcohol intake, kidney health, chronic stress, low dietary intake and certain medications.
When researchers applied this score across 48 studies, a clear pattern emerged: People with chronically low magnesium had higher rates of chronic disease and earlier mortality.
This isn’t because magnesium is a miracle nutrient. It is because low levels disrupt hundreds of biological processes simultaneously.
Magnesium, chronic disease and longevity
According to the review, long-term low magnesium is linked with:
- Cardiovascular disease
- Metabolic syndrome
- Type 2 diabetes
- Osteoporosis
- Chronic, low-grade inflammation
That final point is key. Persistent, low-level inflammation is widely recognised as a driver of accelerated ageing. Magnesium appears to influence these inflammatory pathways directly.
Magnesium and brain ageing
Emerging research also explores magnesium’s role in cognitive health. A 2024 study in the Journal of Neurorestoratology found an association between low magnesium and increased dementia risk. Magnesium supports neuronal communication, protects the blood brain barrier and reduces stress-driven inflammation, all critical elements in healthy cognitive ageing.
Are UK adults getting enough magnesium?
According to the National Diet and Nutrition Survey, a significant proportion of UK adults, particularly women, are not consistently meeting recommended magnesium intakes. Depending on age and dietary patterns, somewhere between 20 to 50 percent of adults fall short.
This does not always lead to clinical deficiency, but it does mean many people live in a suboptimal state that can influence energy, sleep, hormonal balance, metabolic health and overall resilience.
Certain factors further increase magnesium needs:
- Gastrointestinal disorders
- Diabetes
- Thyroid conditions
- Stress
- High caffeine intake
- Alcohol
- Low vitamin D, B6 and selenium
- Diuretics, calcium supplements and laxative use
Does food still contain magnesium?
Yes, but not always at the levels people assume.
Modern farming practices, intensive soil use and refined food production all contribute to lower magnesium content in certain crops compared with historical levels. Foods still contain magnesium, but the baseline has shifted and many people do not eat enough magnesium-rich foods daily to compensate.
Is it worth testing magnesium levels?
Testing magnesium sounds reassuring, but it is not straightforward. Only around 1 percent of the body’s magnesium is found in the blood, with the rest stored in cells, bone and soft tissue. This means serum magnesium often appears normal even when functional levels are low.
From a clinical perspective, symptoms, diet quality, stress load, caffeine intake, alcohol consumption and medication history usually give a clearer picture of magnesium status than a standard blood test. This is why many people with fatigue, poor sleep, muscle tension or increased stress sensitivity benefit from magnesium support despite having normal results on paper.
Signs of low magnesium
Early symptoms can be subtle:
- Fatigue
- Low appetite
- Nausea
- Weakness
Longer-term low levels may contribute to:
- Muscle cramps
- Tingling or numbness
- Higher stress sensitivity
- Palpitations
- Difficulty winding down at night
- Broken sleep patterns
These symptoms often build slowly, which is why magnesium deserves more attention in the context of long-term health.
Magnesium sources
Magnesium-rich foods remain an important part of daily intake:
- Nuts and seeds, especially pumpkin seeds
- Whole grains
- Beans and legumes
- Leafy greens
- Avocado and bananas
- Dairy
But given modern dietary patterns, stress levels and soil variability, many people still fall short even when eating well.
Functional medicine approach: when supplementation helps
Magnesium glycinate is my preferred form for most people. It is well absorbed, gentle on the gut and supportive for sleep, nervous-system regulation and muscle relaxation. Taking it in the evenings is a simple, effective way to support steady magnesium levels, especially during periods of stress, disrupted sleep or higher training load.
Magnesium will not fix everything, but steady levels act like biological scaffolding. They support systems that influence how we feel now and how we age long term.
If you are unsure about your magnesium status, speak with a practitioner, consider your symptoms and overall patterns and adjust your intake accordingly.
Long-term health is not built from dramatic interventions. It is built from steady, nutrient-driven biology, and magnesium sits right at the centre of that.
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by Rebecca Prade | Jan 26, 2026 | WELLBEING
What your appetite and energy levels are actually telling you
Every January, I see the same pattern in clinic. People start the year eating “cleaner,” training harder and drinking less, yet many feel hungrier, flatter, more fatigued and more preoccupied with food than they did only a few weeks earlier. Energy dips creep in mid-morning and mid-afternoon. Cravings get louder, not quieter.
This is often treated as a motivation problem or a lack of discipline. From a functional medicine perspective, that interpretation is almost always wrong.
Cravings and energy dips are rarely random. They are signals coming from the same underlying systems: blood sugar regulation, gut function, and appetite signalling. When these systems are out of sync, the body compensates in predictable ways. If you don’t understand that physiology, it’s easy to end up fighting your own biology and wondering why it keeps pushing back.
This article explains why cravings and low energy so often travel together, how digestion and fibre influence fullness and energy stability, why calorie restriction amplifies both hunger and fatigue, and how supporting the gut helps appetite and energy feel steadier over time.
Cravings are often blood sugar signals, not character flaws
One of the most common drivers of both cravings and low energy is unstable blood sugar. When a meal raises blood glucose quickly, insulin is released to move that glucose into cells. If that rise is sharp, the drop that follows can be sharp too. That drop doesn’t just trigger hunger; it also reduces the brain’s immediate energy supply.
This is what many people experience as sudden hunger, irritability, brain fog, and strong cravings for sugar, refined carbohydrates, or caffeine. The craving itself isn’t for a specific food. It’s a request for fast energy because glucose availability has fallen too quickly.
This is why someone can eat what looks like “enough” on paper and still feel flat, unfocused, and desperate for food two hours later. The body isn’t confused. It’s responding to a predictable dip in glucose availability.
Meals that lack sufficient protein and fibre, or are very low in fat, digest quickly, spike blood sugar, and set up a rebound drop. Repeating this cycle trains the nervous system to expect volatility. Appetite becomes louder, and energy becomes less reliable. Maintaining stable blood glucose levels is one of the least glamorous but most powerful regulators of both appetite and energy that we have.
Energy dips are not a separate issue from appetite
Low energy is often treated as its own problem, managed with more coffee, supplements, or motivation. In reality, energy regulation and appetite regulation are tightly linked. When blood sugar is unstable, the brain experiences shortfalls in fuel availability. This shows up as mental fatigue, reduced concentration, and the familiar mid-afternoon slump.
The body then seeks quick energy sources to compensate. That is why energy dips so often precede cravings. Stabilising blood sugar through adequate protein, fibre, and appropriately timed meals doesn’t just reduce hunger. It improves cognitive energy, mood stability, and the ability to get through the day without constantly reaching for stimulation.
Digestion, fibre, and satiety are often overlooked drivers of both fullness and energy.
Appetite is not only controlled by how much you eat. It is shaped by how well your digestive system processes what you eat. Dietary fibre is not broken down by human digestive enzymes. Instead, it passes through the stomach and small intestine largely unchanged and reaches the colon, where it is fermented by gut bacteria.
That fermentation produces short-chain fatty acids, which play a central role in gut–brain communication. These compounds influence satiety hormones such as GLP-1, slow gastric emptying, and support steadier post-meal energy levels. This is what makes fullness feel settled rather than forced.
When digestion is compromised, these signals weaken. Common contributors include low stomach acid, rushed or distracted eating, chronic psychological stress, and gut inflammation or dysbiosis. In these cases, people often report feeling physically full but mentally unsatisfied. They finish a meal and still want something else, often something stimulating. This is not a restraint issue. It reflects incomplete digestive and satiety signalling.
Appetite signalling, GLP-1, and gut health are tightly linked
GLP-1 is a hormone the body already produces in response to food, particularly protein and fibre. Its roles include slowing stomach emptying, enhancing satiety, supporting insulin release in a regulated way, and helping stabilise post-meal energy.
Crucially, GLP-1 signalling depends on gut health. A gut that is inflamed, under-fuelled, or chronically stressed does not send clean appetite or energy signals. This is why suppressing appetite without addressing digestion tends to fail over time. The signalling pathway itself is compromised.
Why calorie restriction reliably worsens cravings and fatigue
From a physiological standpoint, calorie restriction is interpreted as threat. When energy intake drops too low, particularly alongside increased exercise or psychological stress, the body compensates by increasing hunger hormones, reducing metabolic output, and conserving energy at a cellular level.
This is why ‘being good all day’ so often ends in evening cravings and exhaustion. It is a compensatory response to perceived energy shortage. January-style restriction tends to worsen blood sugar swings, increase reliance on stimulants, and amplify energy crashes. None of this reflects a lack of discipline. It is adaptive biology doing its job.
How supporting the gut steadies appetite and energy over time
When the gut is functioning well, several changes happen quietly but consistently. Meals feel more satisfying. Energy is steadier across the day. Cravings are less urgent and less frequent. Appetite feels responsive rather than reactive.
This does not require perfection. It requires shifting the focus away from control and towards regulation. Supporting the gut often means slowing meals down, spacing eating rather than grazing, prioritising foods that digest well for you, and using targeted support where appropriate rather than blanket restriction.
Most people notice the mental shift first. There is less food noise, fewer crashes, and more sustained energy between meals. Weight changes, if they occur, come later. They are a downstream effect, not the primary mechanism.
A more useful January lens
Cravings are not something to white-knuckle your way through. They are information. Most of the time, they point towards unstable blood sugar, under-fuelled mornings, compromised digestion, or a gut that needs support rather than suppression.
January does not need more discipline. It needs better physiology. When appetite and energy are regulated rather than suppressed, consistency stops feeling like effort and starts feeling normal. That is where sustainable change begins.
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